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Central Surgical Association| Volume 82, ISSUE 3, P337-341, September 1977

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Hepatorenal syndrome: Reversal by peritoneovenous shunt

  • William D. Fullen
    Correspondence
    Reprint requests: William D. Fullen, M.D., Department of Surgery, University of Cincinnati Medical Center, Cincinnati, OH 45267.
    Affiliations
    From the Department of Surgery, University of Cincinnati Medical Center, Cincinnati, Ohio U.S.A.
    Search for articles by this author
DOI:https://doi.org/10.5555/uri:pii:0039606077901428
Hepatorenal syndrome: Reversal by peritoneovenous shunt
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      Abstract

      Two patients with hepatorenal syndrome were treated by insertion of a peritoneovenous shunt. The renal deficit was corrected rapidly in both cases. A 62-year-old woman with a slow onset syndrome with urine output of 100 to 150 ml/day and urinary sodium excretion of 1 mEq/day responded with large volume urinary output and sodium excretion. She is alive with minimal ascites 18 months after shunt. A 53-year-old man with severe nutritional cirrhosis, alcoholic hepatitis, and eventual massive necrosis was treated for bleeding esophageal varices by portacaval shunt. Postoperative massive ascites progressed to acute hepatorenal syndrome. Insertion of a peritoneovenous shunt reversed the renal deficit. He eventually exsanguinated due to a hemorrhagic diathesis caused by massive hepatic necrosis.
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      Article info

      Footnotes

      ☆Presented at the Thirty-fourth Annual Meeting of the Central Surgical Association, Buffalo, N. Y., March 3–5 1977.

      Identification

      DOI: https://doi.org/10.5555/uri:pii:0039606077901428

      Copyright

      © 1977 Published by Elsevier Inc.

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